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A simple finger-prick blood test could help prevent unnecessary prescribing of antibiotics for people with the lung condition chronic obstructive pulmonary disease (COPD), according to a new study.
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A study of more than 300,000 people has found that exposure to outdoor air pollution is linked to decreased lung function and an increased risk of developing COPD. Lung function normally declines as we age, but the new research suggests that air pollution may contribute to the ageing process and adds to the evidence that breathing in polluted air harms the lungs.
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Good heart and lung (cardiorespiratory) fitness in middle age is associated with a lower long term risk of chronic lung disease (COPD), suggests new research.
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More than 1 in 10 people with a range of non-cancerous lung diseases may be sick as a result of inhaling vapors, gas, dust or fumes at work, according to a new statement.
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A global study shows people born very preterm or with very low birthweight have a high risk of lung disease and are not reaching their full airway capacity by early adulthood.
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New research shows that the asthma drug benralizumab failed to decrease annual COPD exacerbation rates for patients with moderate to very severe COPD, a history of frequent moderate and/or severe exacerbations, and eosinophilic inflammation.
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Landmark study confirms ability of non-invasive imaging technique to identify early signs of chronic obstructive pulmonary disease (COPD).
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Researchers have identified proteins that control mucous production and suggest clues to treating colon and airway diseases.
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Researchers revealed that the immune cells basophils caused emphysema in mice with COPD-like features induced by intranasal administration of elastase. They showed that basophils, previously linked mainly to allergies and fighting parasites, initiated a cascade of reactions eventually leading to the release of excess MMP-12 and the destruction of alveolar walls. The team hopes that the findings will lead to breakthroughs in the treatment of COPD.
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Researchers have found a novel, pathogenic entity that is a fundamental link between chronic inflammation and tissue destruction in lungs of patients with COPD. These exosomes from activated neutrophils caused COPD damage when they were instilled into the lungs of healthy mice. Remarkably, neutrophil exosomes from the lung fluids of human patients with COPD and neonates with bronchopulmonary dysplasia also caused COPD lung damage when put into the lungs of healthy mice.

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